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	<title>Cheney Research &#187; NADPH</title>
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		<title>More on Vitamin D3 and CFS</title>
		<link>http://www.cheneyresearch.com/2009/12/more-on-vitamin-d3-and-cfs</link>
		<comments>http://www.cheneyresearch.com/2009/12/more-on-vitamin-d3-and-cfs#comments</comments>
		<pubDate>Thu, 24 Dec 2009 01:24:41 +0000</pubDate>
		<dc:creator>pcheney</dc:creator>
				<category><![CDATA[Hormones in CFS]]></category>
		<category><![CDATA[azoles]]></category>
		<category><![CDATA[Calcium]]></category>
		<category><![CDATA[NADPH]]></category>
		<category><![CDATA[NF Kappa B]]></category>
		<category><![CDATA[P450]]></category>
		<category><![CDATA[Vitamin D3]]></category>

		<guid isPermaLink="false">http://www.cheneyresearch.com/?p=512</guid>
		<description><![CDATA[Vitamin D3 is regulated by P450 enzyme systems that are in turn decoupled due to low NADPH levels in CFS.  This raises important questions regarding the reasons that may underlie low D3 levels typical for most CFS cases.  The finding of increased intracellular calcium by UK investigators may also play into the reasons for finding low D3 in CFS.  Given these deeper issues that may underlie D3 levels suggests that aggressive D3 therapy may not be the best course of action in CFS.  D3 is a highly regulated pro-hormone and there could be good reasons for it to be down-regulated in CFS.]]></description>
			<content:encoded><![CDATA[<p>The effect of vitamin D3 on the echocardiograph ETM in CFS, but not in controls, shows a transient and negative IVRT backflash.  I would not call this effect an &#8220;oxygen toxic effect&#8221; but rather a negative redox shift caused </p>
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		<title>Anabolism vs. Catabolism &#8211; an issue of timing</title>
		<link>http://www.cheneyresearch.com/2009/09/anabolism-vs-catabolism-an-issue-of-timing</link>
		<comments>http://www.cheneyresearch.com/2009/09/anabolism-vs-catabolism-an-issue-of-timing#comments</comments>
		<pubDate>Thu, 17 Sep 2009 22:42:11 +0000</pubDate>
		<dc:creator>pcheney</dc:creator>
				<category><![CDATA[Treatment]]></category>
		<category><![CDATA[anabolism]]></category>
		<category><![CDATA[ATP]]></category>
		<category><![CDATA[catabolism]]></category>
		<category><![CDATA[CSF]]></category>
		<category><![CDATA[D-Ribose]]></category>
		<category><![CDATA[grok]]></category>
		<category><![CDATA[NADPH]]></category>

		<guid isPermaLink="false">https://www.cheneyresearch.com/?p=300</guid>
		<description><![CDATA[When deciding when to use the CSF mixtures, hereafter called the Catabolic CSF Mixture (Heart, Brain and Pancreas) and the Anabolic CSF Mixture (Pancreas, Liver and Kidney), it is important to understand or “grok” your own catabolic period and your own anabolic period of the day. ]]></description>
			<content:encoded><![CDATA[<p>When deciding when to use the CSF mixtures, hereafter called the Catabolic CSF Mixture (Heart, Brain and Pancreas) and the Anabolic CSF Mixture (Pancreas, Liver and Kidney), it is important to understand or “grok” your own catabolic period and your </p>
]]></content:encoded>
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		<title>Catabolism and Anabolism in respect to hormones</title>
		<link>http://www.cheneyresearch.com/2009/05/catabolism-and-anabolism</link>
		<comments>http://www.cheneyresearch.com/2009/05/catabolism-and-anabolism#comments</comments>
		<pubDate>Sat, 02 May 2009 13:36:26 +0000</pubDate>
		<dc:creator>pcheney</dc:creator>
				<category><![CDATA[Hormones in CFS]]></category>
		<category><![CDATA[anabolism]]></category>
		<category><![CDATA[catabolism]]></category>
		<category><![CDATA[DHEA]]></category>
		<category><![CDATA[estrogen]]></category>
		<category><![CDATA[GSH]]></category>
		<category><![CDATA[Melatonin]]></category>
		<category><![CDATA[NADPH]]></category>
		<category><![CDATA[progesterone]]></category>
		<category><![CDATA[vitamin D]]></category>

		<guid isPermaLink="false">http://cheneyresearch.com/?p=84</guid>
		<description><![CDATA[What is so interesting in CFS is that the body realizes that in favoring "catabolism" requires that there be immunosuppression lest there be too much oxidative stress and when favoring "anabolism" there can be immunactivation as the oxidative stress is under better control if you combine anabolism (aka NADPH and increased GSH) with immunoactivation.  The exceptions appear to be estrogen and progesterone, at least on ETM, and my guess is that they are not as catabolically or anabolically  active as Cortisol and DHEA, respectively, but are very immunoactive.  What is not as clear to me is whether Vitamin D is "catabolic" and Melatonin is "anabolic".]]></description>
			<content:encoded><![CDATA[<p>I enjoyed reading the M. Cutolo Review article about melatonin being contraindicated in rheumatoid arthritis (<em>Annals of the Rheumatic Diseases</em> 2005;<strong>64</strong>:1109-1111; doi:10.1136/ard.2005.038588).  What interests me about this is that &#8220;catabolic&#8221; hormones such as cortisol and progesterone, are broadly immunosuppressive whereas </p>
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