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	<title>Cheney Research &#187; estrogen</title>
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		<title>Catabolism and Anabolism in respect to hormones</title>
		<link>http://www.cheneyresearch.com/2009/05/catabolism-and-anabolism</link>
		<comments>http://www.cheneyresearch.com/2009/05/catabolism-and-anabolism#comments</comments>
		<pubDate>Sat, 02 May 2009 13:36:26 +0000</pubDate>
		<dc:creator>pcheney</dc:creator>
				<category><![CDATA[Hormones in CFS]]></category>
		<category><![CDATA[anabolism]]></category>
		<category><![CDATA[catabolism]]></category>
		<category><![CDATA[DHEA]]></category>
		<category><![CDATA[estrogen]]></category>
		<category><![CDATA[GSH]]></category>
		<category><![CDATA[Melatonin]]></category>
		<category><![CDATA[NADPH]]></category>
		<category><![CDATA[progesterone]]></category>
		<category><![CDATA[vitamin D]]></category>

		<guid isPermaLink="false">http://cheneyresearch.com/?p=84</guid>
		<description><![CDATA[What is so interesting in CFS is that the body realizes that in favoring "catabolism" requires that there be immunosuppression lest there be too much oxidative stress and when favoring "anabolism" there can be immunactivation as the oxidative stress is under better control if you combine anabolism (aka NADPH and increased GSH) with immunoactivation.  The exceptions appear to be estrogen and progesterone, at least on ETM, and my guess is that they are not as catabolically or anabolically  active as Cortisol and DHEA, respectively, but are very immunoactive.  What is not as clear to me is whether Vitamin D is "catabolic" and Melatonin is "anabolic".]]></description>
			<content:encoded><![CDATA[<p>I enjoyed reading the M. Cutolo Review article about melatonin being contraindicated in rheumatoid arthritis (<em>Annals of the Rheumatic Diseases</em> 2005;<strong>64</strong>:1109-1111; doi:10.1136/ard.2005.038588).  What interests me about this is that &#8220;catabolic&#8221; hormones such as cortisol and progesterone, are broadly immunosuppressive whereas </p>
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