November 20th, 2009 in New Thinking
I placed three cell phones on his chest wall as he lay on the echocardiograph table and called my office number and left the phones on his mid-sternum for one minute while they rang that number. I then measured the IVRT response every minute after turning the phones off. He “experienced” the documented free energy decline by IVRT criteria as feeling heaviness in his chest. Below is the powerpoint slide of the resulting IVRT decline at each minute after the cell phones were turned off.
October 10th, 2009 in XMRV
There are ways that retroviruses can impact the cell the I believe are especially important to CFS and data exists to support these assertions both in the literature (see below) and in such findings as low SOD and GPx in CFS and provides a mechanism to explain oxygen toxicity by ETM which is nearly universal in CFS.
October 9th, 2009 in XMRV
Proper buffering of the redox set point for the human body’s biological terrain is critical to control intracellular viral replication. Oxidative stess will potentially amplify XMRV replication. The biggest amplifier of oxidative stress due to any cause is NF Kappa B and one of the best inhibitors of NF-kB are the artemisins (Artesunate and Wormwood). Artemisins are also thought to be useful in cancer. We will be exploring the dose response curve for Artemisins to inhibit infectious XMRV in the near future to determine the best dose. We already know that activating NF-kB activates this virus and suppressing NF-kB inhibits XMRV. Artesunate is also known to inhibit HIV and all the associated herpes viruses that are co-factors in the evolution of AIDS and CFS as well.